nf kb activation hok 16b cells (Cell Signaling Technology Inc)
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Cell Signaling Technology Inc
nf kb activation hok 16b cells
Nf Kb Activation Hok 16b Cells, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 92/100, based on 4 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/nf+kb+activation+hok+16b+cells/SignalSilence+SP1+siRNA+II/pm24296811-160-2-42
Average 92 stars, based on 4 article reviews
Nf Kb Activation Hok 16b Cells, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 92/100, based on 4 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/nf+kb+activation+hok+16b+cells/SignalSilence+SP1+siRNA+II/pm24296811-160-2-42
Average 92 stars, based on 4 article reviews
nf kb activation hok 16b cells - by Bioz Stars,
2026-09
92/100 stars
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Activation Assay:Article Title: Mechanisms of IL-8 suppression by Treponema denticola in gingival epithelial cells. Article Snippet: The purpose of this study was to investigate the mechanism(s) of interleukin (IL)-8 suppression by Treponema denticola, one of the major periodontal pathogens, in gingival epithelial cells.. Immortalized human gingival epithelial HOK-16B cells were infected with wild-type (WT), dentilisin-deficient (K1) or flagellin-deficient (flgE) T. denticola in the presence or absence of 2% human serum for 24 h. The levels of IL-8 expression were measured with real-time reverse transcription PCR and ELISA.. In the absence of human serum, the WT and flgE, but not K1, substantially reduced not only the levels of IL-8 protein but also of IL-8 mRNA. Lysis:Article Title: Mechanisms of IL-8 suppression by Treponema denticola in gingival epithelial cells. Article Snippet: The purpose of this study was to investigate the mechanism(s) of interleukin (IL)-8 suppression by Treponema denticola, one of the major periodontal pathogens, in gingival epithelial cells.. Immortalized human gingival epithelial HOK-16B cells were infected with wild-type (WT), dentilisin-deficient (K1) or flagellin-deficient (flgE) T. denticola in the presence or absence of 2% human serum for 24 h. The levels of IL-8 expression were measured with real-time reverse transcription PCR and ELISA.. In the absence of human serum, the WT and flgE, but not K1, substantially reduced not only the levels of IL-8 protein but also of IL-8 mRNA. |